An intricate interaction of numerous structures in the wrist and hand is necessary to produce the remarkable dexterity and precision that characterizes this joint complex.15 The entire upper limb is apparently subservient to the hand in its use as a tactile organ, a means of expression, and a weapon. The study of the hand is inseparable from that of the wrist and the forearm, which function as a single physiologic unit, with the wrist being the key joint.3 The wrist serves as a flexible spacer between the forearm and hand and is critical in setting the length-tension relationship for the extrinsic finger muscles. By far the most important musculoskeletal function of the hand is its ability to grasp objects. However the hand is also an important sensory organ (kinesthetic) and helps to express emotion though gestures, touch, and art. Because of it biomechanical complexity, the function of the hand involves a disproportionately large region of the cortex of the brain. Therefore, dysfunction of the hand can create equally disproportionate disabilities. The hand is the main manipulative organ of the body, performing many different types of functions, and it should not be overlooked in the evaluation for dysfunction.
Interestingly, although the ulna plays a highly significant role in the function of the elbow, it is secondary in the wrist, whereas the radius, having a secondary role in the elbow, has a dominant part in the wrist. The radius flares to become much larger at the distal end, terminating with a lateral extension, called the radial styloid process. The distal end of the ulna also ends in a styloid process, but it is much smaller in comparison with the radial styloid. The distal aspects of the radius and ulna form an articulation with the proximal row of carpal bones directly in the radius and indirectly via an intracapsular disc in the ulna. The eight carpal bones that make up the wrist are arranged in two rows that greatly enhance the hand’s mobility. The proximal row consists of the scaphoid, lunate, triquetrum, and pisiform (in order from medial to lateral). The pisiform overlies the triquetrum, which forms an articulation with the proximal ulna via the interarticular disc. The scaphoid and lunate articulate directly with the radius. The distal row of carpals consists of the trapezium, trapezoid, capitate, and hamate (in order from lateral to medial). The proximal and distal rows of carpal bones collectively form an intercarpal joint, although some movement also occurs between the individual carpal bones (Figure 6-102).
The base of each of the five metacarpals articulates with the distal row of carpals. Five proximal phalanges articulate with each of the metacarpals, followed by a middle and distal phalanx for each of the fingers and a distal phalanx for the thumb.
The numerous ligaments of the wrist, many of which are unnamed, are not all separate entities. They form a crisscross pattern of connections between the radius and ulna to the carpals, between the carpals, from the carpals to the metacarpals, and between the metacarpals (Figure 6-103). The volar radiocarpal and radioulnar carpal ligaments strengthen the joint capsule and wrist anteriorly, while the dorsal radiocarpal ligament provides support posteriorly (Figure 6-104). Radial collateral and ulnar collateral ligaments stabilize the wrist laterally and medially, respectively. Collateral ligaments also support the MP and the interphalangeal joints (Figure 6-105).
Extrinsic and intrinsic muscles function for the wrist and hand (Table 6-10). The wrist flexors and extensors are located in the forearm, attached to the epicondyles of the humerus. As the muscles head distally, their tendons are enclosed in sheaths that offer a smooth environment for sliding. Intrinsic muscles include the interosseous and lumbricales muscles, as well as those responsible for the movements of the thumb and little finger. Six passageways transport the extensor tendons through fibro-osseous tunnels. Fibrous bands running from the retinaculum to the carpal bones form the tunnels (Figure 6-106). The flexor retinaculum spans the scaphoid, trapezium, hamate, and pisiform. It forms a tunnel out of the carpal arch to allow passage of the median nerve and the flexor tendons (Figure 6-107).
TABLE 6-10 Actions of the Muscles of the Wrist and Hand
Actions | Muscles |
---|---|
Wrist flexion | Flexor carpi radialis, abductor pollicis longus, palmaris longus, flexor pollicis longus, flexor carpi ulnaris, and flexor digitorum superficialis and profundus |
Wrist extension | Extensor carpi radialis, extensor digitorum, extensor carpi ulnaris, and extensor pollicis longus |
Wrist adduction (ulnar deviation) | Extensor carpi ulnaris and flexor carpi ulnaris |
Wrist abduction (radial deviation) | Extensor carpi radialis, abductor pollicis longus, and extensor pollicis longus and brevis |
Finger flexion | Flexor digitorum superficialis and profundus |
Finger extension | Extensor digitorum, extensor digiti minimi, and extensor indicis |
Finger abduction | Interosseous muscles |
The complex movements of the wrist are accomplished by the distal radioulnar joint, the radiocarpal joint, and the midcarpal joint. The radiocarpal and midcarpal joints produce the motion at the wrist joint. Wrist flexion and extension, as well as radial and ulnar deviation, are thought to occur around an axis of movement that passes through the capitate (Figure 6-108). However, the multiplicity of wrist articulations and the complexity of joint motion make it difficult to calculate the precise instantaneous axis of motion.12 The close-packed position for the wrist is full extension (Table 6-11). The wrist can undergo approximately 160 degrees of flexion and extension, with extension being slightly greater. Radial and ulnar deviation are possible to 60 degrees, and ulnar deviation is almost twice as great as radial deviation (Figure 6-109). Radial deviation is limited by contact of the scaphoid against the radial styloid process. ROMs for the wrist and hand are listed in Table 6-12.
Figure 6-108 Most wrist flexion occurs at the intercarpal joint, and most wrist extension occurs in the radiocarpal joint.
TABLE 6-11 Close-Packed and Loose-Packed (Rest) Positions for the Wrist and Hand Joints
Closed-Packed Position | Loose-Packed Position | |
---|---|---|
Wrist | Full dorsiflexion | Palmar flexion with slight ulnar deviation |
Hand | Full extension | Flexion with slight ulnar deviation |
Figure 6-109 Dorsal view of the right wrist. A, With ulnar deviation, some extension of proximal carpals occurs. B, With radial deviation, some flexion of the proximal carpal occurs. C, Capitate; H, hamate; L, lunate; S, scaphoid; TP, trapezium; TQ, triquetrum; TZ, trapezoid.
TABLE 6-12 Arthrokinematic and Osteokinematic Movements of the Wrist and Hand Joints
Osteokinematic Movements | Degrees | Arthrokinematic Movements |
---|---|---|
Wrist flexion | 80 | Roll and glide |
Wrist extension | 70 | Roll and glide |
Ulnar deviation | 30 | Roll and glide |
Radial deviation | 20 | Roll and glide |
MCP flexion | 90 | Roll and glide |
MCP extension | 30–45 | Roll and glide |
PIP flexion | 100 | Roll and glide |
PIP extension | 0 | Roll and glide |
DIP flexion | 90 | Roll and glide |
DIP extension | 10 | Roll and glide |
Finger abduction | 20 | Roll and glide |
DIP, Distal interphalangeal joint; MCP, metacarpophalangeal joint; PIP, proximal interphalangeal joint.
With dorsiflexion of the wrist, a supinatory rotation of the carpal bones also occurs, which is mostly a result of the scaphoid moving more with respect to the radius while the lunate and triquetrum relate to the ulna. Furthermore, when moving from flexion to dorsiflexion, the distal row of carpals becomes close-packed with respect to the scaphoid first. This results in the scaphoid moving with the distal row into dorsiflexion, necessitating movement between the scaphoid and lunate as full dorsiflexion is approached. With extension of the wrist, the proximal row of carpals rolls and glides anteriorly with respect to the radius and ulna, and the distal row of carpals moves similarly with respect to the proximal row of carpals. The converse is true of wrist flexion; the proximal row moves posteriorly relative to the radius and ulna as the proximal row moves posteriorly relative to the proximal row of carpals.
Radial and ulnar deviation involve rotary movements between the proximal row of carpals and the radius, as well as between the proximal row and distal row of carpals. Moreover, during radial deviation, the proximal row combines pronation, flexion, and ulnar glide movements with respect to the radius as the distal row combines supination, extension, and ulnar glide movements with respect to the proximal row. Ulnar deviation has the opposite movements.3,16
The hand must be able to change its shape to grasp objects. Three physiologic functional arches running in different directions provide the means for the wrist and hand to conform to a position for grasping (Figure 6-110). Transversely, an arch through the carpal region corresponds to the concavity of the wrist, and distally, the metacarpal arch is formed by the metacarpal heads. Longitudinal arches are formed along each finger by the corresponding metacarpal bone and phalanges. Obliquely, arches are formed by the thumb during opposition with the other fingers. These arches allow coordinating synergistic digital flexion and opposition of the thumb and little finger.
Figure 6-110 The three physiologic arches of the wrist and hand.
(Modified from Nordin M, Frankel VH: Basic biomechanics of the musculoskeletal system, ed 2, Philadelphia, 1989, Lea & Febiger.)
The wrist provides a stable base for the hand, and its position controls the length of the extrinsic muscles to the digits. The muscles stabilize the wrist, as well as provide for the fine movements of the hand, to place it in its functioning position. The positioning of the wrist has a significant influence on the strength of the fingers. For most effective action of the extrinsic muscles of the fingers, the wrist usually must move in a direction opposite the movement of the fingers.
The naturally assumed position of the hand to grasp an object, or the position from which optimal function is most likely to occur, is termed the functional position (Figure 6-111). The functional position occurs when the wrist is extended 20 degrees and the ulna is deviated 10 degrees, the fingers are flexed at all of their joints, and the thumb is in a midrange position, with the MP joint moderately flexed and the interphalangeal joints slightly flexed. Prehensile functions of the hand are unique and fundamental characteristics (Figure 6-112).
The wrist and hand are prone to injuries from trauma, commonly a fall on the outstretched hand. The radial side of the wrist and hand tends to take the majority of the force from such a trauma. Displacement, instability, and rotary subluxation of the scaphoid often occur, creating pain on dorsiflexion of the wrist and limited ROM. Palpable tenderness will be present over the joint space between the scaphoid and the lunate. Occasionally, the scaphoid can be felt slipping as the wrist is moved, or a painful click may be perceived. A space of more than 3 mm between the scaphoid and lunate may be seen on a closed-fist supinated radiographic view of the wrist.
Trauma to an outstretched hand that is forcefully flexed or extended may cause a fracture to the radius. A Colles fracture occurs when the wrist is in dorsiflexion and the forearm is in pronation. Local pain and tenderness to palpation and vibration are important physical findings; however, the radiograph is the most important tool for determining the presence of a fracture. Manipulative therapy to this area is then contraindicated.
Singular trauma, such as a fall, or repetitive activities may lead to a sprain of the ligaments of the wrist. Moreover, when the wrist is subjected to sudden increases in workload, such as in gripping or lifting, or racquet games that require flexion and extension of the wrist, the tendons crossing the wrist can become inflamed, resulting in tendinitis. In addition, a possible response to repeated twisting and straining is a localized nodular swelling, called a ganglion. Likely a defense mechanism, the ganglion is characterized by a fibrous outer coat that covers a thick gelatinous fluid derived from the synovium lining the tendon sheaths.
Undoubtedly, the most noted condition affecting the wrist and hand is carpal tunnel syndrome, a peripheral entrapment neuropathy involving the median nerve. The median nerve lies superficial to the flexor tendons beneath the tense transverse carpal ligament (flexor retinaculum), making the carpal tunnel just barely adequate to accommodate these structures. In the act of grasping an object, particularly with the wrist in flexion, the flexor tendons are displaced forward and can compress the nerve against the unyielding ligament. Narrowing of the carpal tunnel can occur through bony deformity after fracture, degenerative joint disease, synovial swelling of a tendon or of the wrist joint ligaments, and thickening of the transverse carpal ligament (Figure 6-113). Most often, however, no definitive local cause for nerve compression can be detected. Moreover, Upton and McComas20 has identified the possibility that peripheral nerves can be compressed at more than one spot along their course, creating “double-,” “triple-,” and “quadruple-crush” syndromes. Therefore, although the patient’s clinical picture may be defined as carpal tunnel syndrome, the nerve compression may not necessarily be at the wrist but may be at the elbow, shoulder, or neck. This syndrome occurs more often in women, with onset commonly between 40 and 50 years of age. Slight paresthesias may precede the onset of the acute symptoms for several months. Then, paroxysms of pain, paresthesia, and numbness occur in the area of the median nerve distribution. The patient is often awakened at night by numbness or pain that can be described as burning, aching, prickly, or pins-and-needles. Motor weakness of the thumb adductor or opposer may be found. The patient may describe relief from dangling the hand over the side of the bed, shaking the hand vigorously, or rubbing it.
Figure 6-113 Cross-section of the wrist, showing the relationship between the carpal bones, tendons, flexor retinaculum, and median nerve.
Because the wrist structures are innervated primarily from segments C6 through C8, lesions affecting structures of similar derivation may refer pain to the wrist and vice versa. Symptoms experienced at the wrist and hand must always be suspected as possibly having a more proximal origin (Figure 6-114).
Figure 6-114 Symptoms in the hand and wrist must be suspected of having a more proximal origin.
(From Magee DJ: Orthopedic Physical Assessment, ed 5, St Louis, 2008, Sauders.)
Observe the wrist and hand for general posture and attitude. In the resting attitude of the hand, the MP and interphalangeal joints are held in a position of slight flexion. Observe the arm and hand for natural swing when the patient walks. Also, note functional activities of the hand and wrist, including the firmness of the person’s handshake, as well as temperature and moisture of the hand. The dominant hand should be determined. Sometimes this can be done by noting the hand with more developed musculature, but is most easily done simply by asking the patient.
To begin the evaluation of the wrist and hand, osseous symmetry, bony relationships, and pain production are identified through static palpation of the wrist and hand (Figure 6-115). Palpate the radius and ulna distally, identifying each of their styloid processes. Just distal to the radial styloid and in the anatomic snuffbox, the scaphoid can be palpated. Wrist flexion will facilitate the palpation of the lunate, which lies next to the scaphoid. The triquetrum and pisiform overlie one another and are just distal to the ulnar styloid. The trapezium can be identified at the base of the first metacarpal. The trapezoid lies at the base of the second metacarpal. The capitate is found between the base of the third metacarpal and the lunate. The hook of the hamate, and hence the hamate, can be found on the palmar surface, just distal and to the thumb side of the pisiform. Then palpate the metacarpals through the palm of the hand, with the fingers along the shaft of the metacarpal on the palmar surface while the thumb is over the dorsal surface. Finally, palpate the 14 phalanges (2 for the thumb and 3 for each finger).
Identify tone, texture, and tenderness changes through soft tissue palpation of the flexor and extensor tendons, the thenar eminence, and the hypothenar eminence. Determine patency of the radial and ulnar arteries (Allen test) and take a radial pulse.
Evaluate accessory joint motions for the wrist and hand articulations to determine the presence of joint dysfunction (Table 6-13). Assess A-P and P-A glide of the distal radioulnar joint with the patient supine or sitting. Grasp the distal radius with one hand and the distal ulna with the other. Apply an opposing A-P and P-A shearing stress between the radius and ulna (Figure 6-116).
TABLE 6-13 Accessory Joint Movements of the Wrist and Hand Joints
Joint | Movement |
---|---|
Distal radioulnar | |
Intercarpal | |
Individual carpals | |
Intermetacarpal | |
Metacarpophalangeal and interphalangeal |
A-P, Anterior-to-posterior; L-M, lateral-to-medial; M-L, medial-to-lateral; P-A, posterior- to-anterior.
Figure 6-116 Assessment of anterior-to-posterior and posterior-to-anterior glide of the distal left radioulnar joint.
Conduct M-L compression of the distal radioulnar joint with the patient supine or sitting, and use both hands to encircle the distal radius and ulna. Use both hands to apply an M-L compression stress to the distal radius and ulna (Figure 6-117).
In evaluating long-axis distraction of the intercarpal joint, the patient can be seated or supine. Grasp the distal forearm with one hand and the distal wrist with the other. While stabilizing the forearm, distract the wrist in the long axis (Figure 6-118).
Perform M-L tilt and glide of the intercarpal joints with the patient seated and the affected arm raised in forward flexion. Stand on the affected side, facing the lateral aspect of the arm. Grasp the distal radius and ulna with your proximal hand while grasping the patient’s distal wrist with your distal hand. Use both hands to create opposing forces, creating a shearing stress (M-L glide) (Figure 6-119) and radial and ulnar deviation stress (M-L tilt) (Figure 6-120).
Figure 6-119 Assessment of medial-to-lateral and lateral-to-medial glide of the left intercarpal joint.
Figure 6-120 Assessment of medial-to-lateral and lateral-to-medial tilt of the left intercarpal joint.
Assess A-P and P-A glide of the intercarpal joint, with the patient seated and arm raised in forward flexion. Stand on the affected side. Grasp the distal radius and ulna with your proximal hand while grasping the patient’s distal wrist with your distal hand. Using both hands to create opposing forces, stress the intercarpal joints in an A-P and P-A glide, looking for a springing joint play movement (Figure 6-121).
Figure 6-121 Assessment of anterior-to-posterior and posterior-to-anterior glide of the left intercarpal joint.
Assess A-P and P-A glide of the individual carpal bones with the patient seated and the affected arm raised in forward flexion. Stand and face the patient. Use your thumb and index or middle fingers to contact the anterior and posterior surfaces of the carpal bone to be evaluated while using your other hand to stabilize the rest of the wrist. Apply an A-P and P-A stress to each individual carpal bone, looking for a springing joint play movement (Figure 6-122, A and B).
Figure 6-122 Assessment of posterior-to-anterior (A) and anterior-to-posterior (B) glide of the individual carpals of the left wrist.
Assess A-P and P-A glide of the intermetacarpal joints with the patient seated and the affected arm raised in forward flexion. Stand and face the patient, grasp the adjacent metacarpals with both hands, and stress them in an A-P and P-A glide (Figure 6-123).
Figure 6-123 Assessment of anterior-to-posterior and posterior-to-anterior glide of the left intermetacarpal joints.
Evaluate the MP and interphalangeal joints in a similar fashion. With the patient seated, grasp the proximal member of the joint to be tested with one hand while grasping the distal member of the joint being tested with the other hand. Then stress each MP or interphalangeal joint with long-axis distraction, A-P and P-A glide, L-M and M-L glide, and internal and external rotation (Figures 6-124 and 6-125).
The application of an impulse thrust often can be performed using the accessory joint motion test procedure and adding the impulse thrust at the end. Although this is true of any joint in the body, fewer adjustive procedures are unique or different from the testing procedure for the wrist and hand. Box 6-8 identifies the adjustive procedures for the joints of the wrist and hand.
BOX 6-8 Wrist and Hand Adjustive Techniques
Figure 6-130 Adjustment for medial-to-lateral and lateral-to-medial glide (A) and medial-to-lateral and lateral-to-medial tilt (B) of the left intercarpal joint.
The hip joint is one of the largest and most stable joints in the body.17 In contrast to the other extremity joints and, specifically, its counterpart in the upper extremity, the shoulder, the hip has intrinsic stability provided by its relatively rigid ball-and-socket configuration (Figure 6-135) and an extensive set of capsular ligaments. Although dysfunction of the hip is not as frequently encountered as dysfunction in the spine and other extremity joints, its identification and treatment are very important and often overlooked. A pathologic or traumatized hip can create a wide range of functional limitations, including difficulty in walking, dressing, driving a car, climbing stairs, and lifting and carrying loads. The hip joint must accommodate the great deal of mobility necessary for gait and the performance of daily activities. Furthermore, the hip joint is a multiaxial articulation that must form a stable link between the lower limb and the spine and pelvis.
The hip is a deep ball-and-socket joint with a spherical convex surface on the head of the femur and a concave articular surface formed by the acetabulum of the pelvis. The acetabulum is formed by a fusion of the three bones that make up the innominate: the ilium (superior), the ischium (posteroinferior), and the pubic bone (anteroinferior) (Figure 6-136). A fibrocartilaginous acetabular labrum surrounds the rim of the acetabulum, effectively deepening it and serving to protect the acetabulum against the impact of the femoral head in forceful movements. Hyaline cartilage lines the horseshoe-shaped surface of the acetabulum. The center of the acetabulum is filled in with a mass of fatty tissue covered by a synovial membrane. The cavity of the acetabulum is directed obliquely anteriorly, laterally, and inferiorly. The inferior component is important because of the transference of weight from the upper body through the sacroiliac joints into the head of the femur and down its shaft.
The femur is the longest bone in the body, as well as one of the strongest. It must withstand not only weight-transmission forces but also those forces developed through muscle contraction. The femoral head is completely covered with hyaline cartilage, except for a small pit near its center, known as the fovea capitis. The cartilage is thicker above and tapers to a thin edge at the circumference.
The femoral neck forms two angular relationships with the femoral shaft that influence hip function (Figure 6-137). The angle of inclination of the femoral neck to the shaft in the frontal plane (the neck-to-shaft angle) is approximately 125 degrees (90 to 135 degrees). This angle offsets the femoral shaft from the pelvis laterally and facilitates the freedom of motion for the hip joint. An angle of more than 125 degrees produces a coxa valga, whereas an angle of less than 125 degrees results in coxa vara. A deviation either way can alter the force relationships of the hip joint. The angle of anteversion is the second angle associated with the femoral neck and is formed as a projection of the long axis of the femoral head and the transverse axis of the femoral condyles. This angle should be approximately 12 degrees but has a great deal of variation, of which 10 to 30 degrees is considered within normal limits. Any increase in this anterior angulation is called excessive anteversion and results in a toe-in posture and gait. An angle that is less than ideal produces a retroversion and an externally rotated leg posture (toe-out) and gait.
Figure 6-137 The proximal femur. A, Coronal section, showing the trabecular patterns and the angle of inclination of the femoral neck. B, Apical view of left femur, showing the angle of anteversion.
The atmospheric pressure holding the head of the femur in the acetabulum is approximately 18 kg. This could support the entire limb without ligamentous or muscular assistance, although capsular ligament and muscular tension do help keep the head of the femur stable in the acetabulum.
A unique trabecular pattern corresponding to the lines of force through the pelvis, hip, and lower extremity are developed through the course of the femoral neck (Figure 6-138). Tension trabeculae are more superior and run from the femoral head to the trochanteric line. Compression trabeculae are inferior and run from the trochanteric area to the femoral head. The epiphyseal plates are at right angles to the tension trabeculae, which likely places them perpendicular to the joint reaction force on the femoral head. Aging produces degenerative changes that gradually cause the trabeculae to resorb, predisposing the femoral neck to fracture.
The hip joint is completely covered by an articular capsule that attaches to the rim of the acetabulum, to the femoral side of the intratrochanteric line, and to parts of the base of the neck and adjacent areas. The joint capsule is a cylindrical structure resembling a sleeve running between its attachment around the peripheral surface of the acetabular labrum to the femoral neck (Figure 6-139). It therefore encloses not only the head of the femur but the neck as well. From its femoral attachments, some of the fibers are reflected upward along the neck as longitudinal bands, termed retinacula. The capsule is thicker toward the upper and anterior part of the joint, where the greatest amount of resistance is required. Some deep fibers of the distal portion of the capsule are circular, coursing around the femoral neck and forming the zona orbicularis. They form somewhat of a sling, or collar, around the neck of the femur.
Figure 6-139 Diagrammatic representation of the cylindrical joint capsule, showing the orientation of fibers to resist stresses.
The joint capsule is reinforced and supported by strong ligaments named for the bony regions to which they are attached (Figure 6-140). The iliofemoral ligament lies anteriorly and superiorly and forms an inverted Y from the lower part of the anteroinferior iliac spine to the trochanteric line of the femur. It prevents posterior tilt of the pelvis during erect standing, limits extension of the hip joint, and is responsible for the so-called “balancing on the ligaments” maneuver that occurs in the absence of muscle contraction.
The ischiofemoral ligament consists of a triangular band of strong fibers extending from the ischium below and behind the acetabulum to blend with the circular fibers of the joint capsule and attaching to the inner surface of the greater trochanter. It reinforces the posterior portion of the capsule and limits excessive medial rotation, abduction, and extension.
The pubofemoral ligament is attached above to the obturator crest and the superior ramus of the pubis, and below it blends with the capsule and with the deep surface of the vertical band of the iliofemoral ligament. It reinforces the medioinferior portion of the joint capsule and limits excessive abduction, lateral rotation, and extension.
The apex of the ligamentum teres attaches to the fovea capitis femoris, and its base attaches by two bands, one into either side of the acetabular notch (Figure 6-141). This ligament does not truly contribute to the support of the joint, although it becomes taut when the thigh is semiflexed and then adducted or externally rotated. It sometimes contains and offers some protection for a nutrient artery that supplies the femoral head. The ligamentum teres is lined with synovium and is believed to play a role in assisting with joint lubrication. As such, it may act somewhat like the meniscus in the knee by spreading a layer of synovial fluid over the articular surface of the head of the femur. The transverse ligament crosses the acetabular notch, converting the notch into a foramen through which the artery that supplies the head of the femur runs.
The hip is supported by strong muscles on all four sides (Table 6-14). The posterior musculature, composed of the gluteus maximus, posterior fibers of the gluteus medius, hamstrings, and piriformis, provides posterior stability for the hip joint. Anterior joint stability is provided by the iliopsoas, sartorius, and rectus femoris muscles. The tensor fascia lata, gluteus medius, and gluteus minimus provide lateral stability. Medial stability comes from the pectineus, adductors, and gracilis muscles. With the amount of movement and soft tissues lying in the different planes associated with the hip, many bursae exist; however, only three have important clinical significance. The iliopectineal bursa lies between the iliopsoas muscle and the hip joint capsule. It sometimes communicates with the joint cavity itself, and excess fluid from trauma may spill into it. The combined motions of hip flexion and adduction or excessive extension can compress the inflamed bursa, creating pain. The trochanteric bursa separates the tendon of the gluteus maximus and the iliotibial band from the greater trochanter. Direct trauma to this area or overuse of the joint may irritate the bursa, causing it to become inflamed. A third bursa, the ischiogluteal bursa, lies superficially over the ischial tuberosity. This bursa will become inflamed in those who sit for prolonged periods, and they need to flex and extend their hips periodically.
TABLE 6-14 Actions of the Muscles of the Hip Joint
Action | Muscles |
---|---|
Extension | Gluteus maximus, gluteus medius, and hamstrings |
Flexion | Iliopsoas, sartorius, rectus femoris, tensor fascia lata, gracilis, and pectineus |
Abduction | Tensor fascia lata, gluteus medius and minimus, and piriformis |
Adduction | Adductors, pectineus, and gracilis |
External rotation | Piriformis, gemelli, obturators, and quadratus femoris |
Internal rotation | Tensor fascia lata, gluteus medius and minimus, and gracilis |
Because of the hip’s role in posture and gait, the combined actions of many powerful muscles are required. Hip flexion is accomplished primarily by the iliopsoas and is assisted by the rectus femoris, pectineus, adductor longus, gracilis, tensor fascia lata, and sartorius. Hip extension occurs through the contraction of the gluteus maximus muscle, easily one of the most powerful muscles in the body. Hip extension is assisted by the hamstrings and the posterior fibers of the gluteus medius. The gluteus medius and minimus, the tensor fascia lata, and to a lesser extent, the piriformis create hip abduction. Hip adduction is the primary role of the adductor muscles, the gracilis, and the pectineus, with some influence from the hamstrings. External rotation of the hip occurs through contraction of the piriformis, obturators, gemelli, and quadratus femoris muscles. Internal rotation of the hip is accomplished by the tensor fascia lata, gluteus medius, gluteus minimus, and gracilis muscles.
Several of the muscles that act at the hip joint also act with equal or greater effectiveness at the knee joint. These are known as two-joint muscles of the lower extremity. The location and line of pull or action of the muscles make it relatively easy to understand the mechanics of testing any individual muscle.
The movements of the femur are similar to those of the humerus but not as free because of the depth of the acetabulum. In the standing position, the shaft of the femur slants somewhat in a medial direction and is not vertically straight. This places the center of motion of the knee joint more nearly under the center of motion of the hip joint. Therefore, the mechanical axis of the femur is almost vertical. The degree of slant of the femoral shaft depends on both the angle between the neck and the shaft and the width of the pelvis. Seen from the side, the shaft of the femur bows forward. These orientations of the femur are provisions for resisting the stresses and strains sustained in walking and jumping and for ensuring proper weight transmission.
Pelvic rotation about the hip accounts for a significant portion of forward bending. Trunk flexion from the erect posture through approximately the first 45 to 60 degrees involves primarily the lumbar spine, with further forward bending occurring because of the pelvis rotating about the hip (Figure 6-142). The iliofemoral and ischiofemoral ligaments twist as they go from the pelvic attachment to the femur. In the erect neutral position, these ligaments are under moderate tension. Thigh extension “winds” these ligaments around the neck of the femur and tightens them. Furthermore, during posterior tilting of the pelvis, these ligaments are taut and therefore are responsible for maintaining optimal pelvic position (Figure 6-143). Anterior hip and thigh pain may occur as a result of tension in these ligaments from excessive posterior pelvic tilting. In contrast, flexion of the hip “unwinds” these ligaments. Moreover, anterior pelvic tilting is not prevented by these ligaments, and the hip extensors must play an important role in stabilizing the pelvis in the anteroposterior direction. The twisting of these ligaments, as well as the twisting that occurs within the joint capsule, draws the joint surfaces into a close-packed position through a “screw-home” movement of the joint surfaces. The close-packed position of the hip is in extension, abduction, and internal rotation (Box 6-9). According to Kapandji,21 erect posture tilts the pelvis posteriorly, relative to the femur, causing these ligaments to become coiled around the femoral neck.
Figure 6-142 Trunk flexion begins with lumbar spine flexion, followed by pelvic flexion at the hip joints.
Figure 6-143 Diagrammatic representation of the effects of flexion and extension on the ischiofemoral and iliofemoral ligament. A, Right hip in the neutral position. B, Extension tightens ligaments. C, Flexion slackens ligaments.
During flexion, a forward movement of the femur occurs in the sagittal plane. If the knee is straight, the movement is restricted by the tension of the hamstrings. In extreme flexion, the pelvis tilt supplements the movement at the hip joint. Extension is a return movement from flexion. Hyperextension, however, is a backward movement of the femur in the sagittal plane. This movement is extremely limited. In most people, this is possible only when the femur is rotated outward. The restricting factor is the iliofemoral ligament at the front of the joint. The advantage of restriction of this movement is that it provides a stable joint for weight-bearing without the need for strong muscular contraction. The movements are mostly rotary actions. Abduction is described as a sideward movement of the femur in the frontal plane, with the thigh moving away from the midline of the body. A greater range of movement is possible when the femur is rotated outward. Adduction is a return movement from abduction, whereas hyperadduction is possible when the other leg is moved out of the way. Abduction and adduction motions are a combination of roll and glide. Internal rotation and external rotation are rotary movements of the femur around its longitudinal axis, resulting in the knee turning inward and outward, respectively (Figure 6-144). Circumduction is a combination of flexion, abduction, extension, and adduction performed sequentially in either direction (Table 6-15).
TABLE 6-15 Arthrokinematic and Osteokinematic Movements of the Hip Joint
Osteokinematic Movements | Degrees | Arthrokinematic Movements |
---|---|---|
Flexion | 120 | Rotation |
Extension | 30 | Rotation |
Abduction | 45–50 | Roll and glide |
Adduction | 20–30 | Roll and glide |
Internal rotation | 35 | Roll and glide |
External rotation | 45 | Roll and glide |
When the hip is externally rotated, the anterior ligaments become taut while the posterior ligaments relax. The converse is true when the hip is internally rotated (Figure 6-145). During adduction the inferior part of the joint capsule becomes slack while the superior portion becomes taut. The opposite is true during abduction; the inferior part of the capsule becomes taut, and the superior portion relaxes and folds on itself (Figure 6-146). During abduction the iliofemoral ligament becomes taut, and the pubofemoral ligament and ischiofemoral ligament slacken. Again, during adduction, the opposite occurs; the pubofemoral ligament and the ischiofemoral ligament become taut, and the iliofemoral ligament slackens.
Figure 6-145 Transverse section through the left hip, demonstrating effects of internal and external rotation on the ischiofemoral, iliofemoral, and pubofemoral ligaments. A, Neutral position. B, External rotation slackens posterior ligaments and stretches anterior ligaments. C, Internal rotation slackens anterior ligaments.
Figure 6-146 Coronal section through the right hip viewed from anterior to posterior, demonstrating the effects of abduction and adduction of the joint capsule. A, Neutral. B, Adduction tightens superior fibers and slackens inferior fibers. C, Abduction tightens inferior fibers and slackens superior fibers.
Pelvic stability in the coronal plane is secured by the simultaneous contraction of the ipsilateral and contralateral adductors and abductors. When these antagonistic actions are properly balanced, the pelvis is stabilized in the position of symmetry (Figure 6-147). If, however, an imbalance exists between the abductors and the adductors, the pelvis will tilt laterally to the side of adductor predominance. If the pelvis is supported by only one limb, stability is provided only by the action of the ipsilateral abductors. An insufficiency in the abductor muscles and, specifically, the gluteus medius results in the body weight not being counterbalanced, resulting in a pelvic tilt to the opposite side. The severity of muscular insufficiency relates directly to the degree of lateral pelvic tilting. Furthermore, during standing on one leg, the femoral head must support more than the weight of the body. The total force acting vertically at the femoral head is equal to the force produced by the pull of the abductors plus the force produced by the body weight or up to three times the body weight.3
Figure 6-147 Pelvic stability in the coronal plane is produced by a balance between the abductors and adductors.
The resting, or loose-packed, position of the hip, or that in which the joint capsule is totally slack, is 10 degrees of flexion, 10 degrees of abduction, and 10 degrees of external rotation. This position will often be assumed to accommodate swelling. Pathomechanical changes and degenerative processes can alter the resting position. A joint posture of flexion, adduction, and external rotation is the classic capsular pattern of the hip.
Although the hip joint exhibits 3 degrees of freedom of motion and is analogous to the glenohumeral joint, the hip is intrinsically a much more stable joint. The hip, however, is still quite prone to pathomechanic changes and, as such, is often overlooked as a source for mechanical joint dysfunction. Clinically, pain originating in the hip joint is primarily perceived as involving the L3 segment, although derivation of the hip joint is from segments L2 to S1. Hip pain can be the result of referral from the facets of the lower lumbar spine. Moreover, the knee also refers pain to the hip area, and the hip can refer pain to the knee (Figure 6-148).
Figure 6-148 Hip pain can be referred from the knee or lumbar spine, and hip disorders can refer pain to the lumbar spine and knee.
The muscles working across the hip joint are subject to strain, either through overuse (chronic strain) or overstress (acute strain or trauma). Tenderness is usually localized to the involved muscle, and the pain increases with resisted contraction. Commonly strained muscles include the sartorius, rectus femoris, iliopsoas, hamstrings, and adductors.
Trochanteric bursitis, a result of overuse or direct injury, presents as pain felt primarily over the lateral hip region, which is often aggravated by going up stairs. The pain is usually described as deep and aching pain that began insidiously. Getting in and out of a car is sometimes listed as a precipitating factor. Point tenderness is found over the inflamed bursa at the posterolateral aspect of the greater trochanter.
Entrapment of several peripheral nerves can occur in association with hip dysfunction. The femoral nerve lies close to the femoral head, and trauma or hematoma may produce entrapment, causing weakness of the hip flexors and local tenderness in the groin. The sciatic nerve, which passes deep to or through the piriformis muscle, may be compressed with contraction of the piriformis muscle. A sciatic radiculopathy with concomitant motor and sensory changes may result. The lateral femoral cutaneous nerve is prone to entrapment near the anterior superior iliac spine (ASIS), where the nerve passes through the lateral end of the inguinal ligament. Entrapment creates a condition called meralgia paresthetica and is characterized by a burning pain in the anterior and lateral portions of the thigh. The condition may be associated with a biomechanical dysfunction of the lumbopelvic complex and postural unleveling of the pelvis.22
Use radiographic examination of children with hip pain (anteroposterior and frog leg) to evaluate the integrity of the capital femoral epiphysis. A slipped capital femoral epiphysis may occur, creating hip or knee pain. On examination, the hip will tend to swing into external rotation instead of flexion. A referral for a surgical consult is indicated.
An unrecognized or improperly treated slipped capital femoral epiphysis or a reactive synovitis may occlude the blood supply to the femoral head, and part or all of the femoral head may die as a result of avascular necrosis. These avascular changes usually involve the superoanterolateral weight-bearing part of the femoral head, and in later stages, this area becomes irregular, collapsed, and sclerotic. Examine radiographs for rarefaction of the femoral head, characteristic of Legg-Calvé-Perthes avascular necrosis.
To begin evaluation of the hip, observe the joint for the presence of any skin lesions associated with trauma, signs of inflammation, and the presence of pelvic obliquity. Observe gait patterns, although usually a pathomechanic hip dysfunction will not be severe enough to create a noticeable change in gait. However, toeing-in or toeing-out may be identified.
Note osseous symmetry and relationships between the greater trochanters, ASIS and posterior superior iliac spine, iliac crests, ischial tuberosities, and pubic symphysis. Identify tone, texture, and tenderness changes through soft tissue palpation of the bursa, inguinal ligament, hip flexors, hip extensors, hip adductors, and hip abductors.
Evaluate accessory joint motions for the hip joint for the presence of joint dysfunction (Box 6-10). Evaluate long-axis distraction of the iliofemoral joint, with the patient supine and the affected side close to the edge of the table. Straddle the patient’s distal thigh, grasping the area just proximal to the epicondyles with your knees. With your outside hand, palpate the greater trochanter while you stabilize the pelvis at the ASIS with your inside hand. By straightening your legs, you can induce a long-axis distraction into the hip joint and perceive a springing joint play movement with the contact on the greater trochanter (Figure 6-149).
Figure 6-149 Referred pain around the hip. Right side demonstrates referral to the hip. Left side shows referral from hip.
(Modified from Magee DJ: Orthopedic physical assessment, ed 5, St Louis, 2008, Saunders.)
Evaluate internal and external rotation, with the patient supine with the affected hip flexed to 90 degrees and the knee flexed to 90 degrees. Stand on the affected side, facing cephalad and using your outside hand to palpate the hip joint and greater trochanter while grasping the patient’s calf and thigh area with your inside arm. Then induce internal and external rotational stresses while evaluating for the presence of a springy end feel–type motion (Figure 6-150).
Determine A-P and P-A glide movement, with the patient supine and the involved leg slightly abducted. Straddle the patient’s thigh just above the knee. Grasp the proximal thigh with both hands and induce an A-P and P-A stress, feeling for the presence of a springing joint play movement (Figure 6-151).
Figure 6-151 Assessment of anterior-to-posterior (A) and posterior-to-anterior (B) glide of the left hip joint.
Evaluate inferior glide of the hip in flexion, with the patient supine and the involved knee flexed to 90 degrees and the hip flexed to 90 degrees. Stand on the involved side, facing the patient and bending over so that the patient’s calf can rest over your shoulder. Grasp the anterior aspect of the proximal thigh and create a caudal stress toward the foot end of the table, evaluating for the presence of a springing end-feel movement (Figure 6-152).
The manipulative techniques used to treat hip disorders aim to restore normal joint mechanics, which will then ideally allow full pain-free functioning of the hip joint. Box 6-11 identifies the adjustive procedures for the hip.
Figure 6-154 Modification for long-axis distraction using a towel. A, Wrapping the towel. B, Grasping the towel to apply the thrust.
The distal end of the femur and the proximal end of the tibia are connected by numerous ligaments and stabilized by strong muscles to form the very complicated knee joint. This joint is situated between the body’s two longest lever arms and therefore must be able to transmit significant loads as it sustains high forces through upright posture and gait. Three articular complexes are typically discussed in conjunction with the knee: the tibiofemoral, patellofemoral, and tibiofibular articulations. However, only the tibiofemoral and patellofemoral articulations participate in knee joint activity. The tibiofibular articulation does not actually contribute to the actions of the knee. Instead it is part of the ankle joint complex, moving with inversion and eversion as well as dorsiflexion and plantar flexion. However, dysfunctional processes in the proximal tibiofibular articulation can affect other knee functions and can be a source of knee pain.
The knee joints are located between the ends of each supporting column of the body and are therefore subjected to severe stress and strain in the combined function of weight-bearing and locomotion. For adaptation to weight-bearing stresses, the knee has large condyles that are padded by the intra-articular menisci. The articular structure allows a wide ROM to facilitate locomotion, and the knee has strong ligaments on its sides to resist lateral stresses. To combat the downward pull of gravity and to meet the demands of violent locomotor activities, such as running and jumping, the knee is provided with powerful musculature. Injuries to the ligaments and cartilage of the knee are two common consequences of the large function demands placed on the knee.23
The femoral shaft lies in oblique alignment with the lower leg, which produces a physiologic valgus angle of approximately 170 to 175 degrees (Figure 6-161). The distal end of the femur is expanded to form a large, convex, U-shaped articular surface (Figure 6-162). The medial and lateral femoral condyles lie on the end of the U shape and are separated by the intercondylar fossa. Anteriorly, the articular surface of the femoral condyles forms the patellar groove. The proximal end of the tibia is flattened to create a plateau with a bifid, nonarticulating intracondylar eminence, dividing the plateau into medial and lateral sections to accommodate the medial and lateral femoral condyles. The tibial tuberosity projects from the anterior surface of the tibia, serving as the point of insertion of the quadriceps tendon (Figure 6-163). The patella, the largest sesamoid bone in the body, lies embedded within the quadriceps tendon. It is triangular in shape, with its apex directed inferiorly. The anterior surface is nearly flat, and a longitudinal ridge divides the posterior surface into medial and lateral articulating facets. The longitudinal ridge fits into the patellar groove of the femur. The proximal head of the fibula is expanded and contains a single facet that corresponds with a facet on the posterolateral aspect of the rim of the tibial condyle.
Internal to the joint are the cruciate ligaments, arranged in a crisscross manner, providing A-P, as well as M-L, stability at the knee. They prevent excessive medial rotation of the tibia and help maintain contact between the articular surfaces of the tibia and femur (Figures 6-164 to 6-167). The anterior cruciate ligament extends from the anterior aspect of the intercondylar eminence of the tibia and runs posteriorly and superiorly to the medial side of the lateral condyle of the femur. The posterior cruciate ligament attaches from the posterior intercondylar eminence of the tibia, extending anteriorly and superiorly to the lateral side of the medial condyle of the femur. The anterior cruciate ligament resists anterior displacement of the tibia and checks extension movements. In contrast, the posterior cruciate ligament primarily checks posterior displacement of the tibia and resists internal rotation of the tibia. The posterior cruciate ligament, lying medial to the anterior cruciate ligament, is the strongest of the knee ligaments. It is especially important for providing M-L stability of the knee when in extension.
Figure 6-167 A, Anterior cruciate ligament becomes taut while in flexion. B, Posterior cruciate ligament becomes taut on knee extension.
The collateral ligaments provide M-L stability and support for the knee while also preventing excessive external rotation of the tibia. The medial, or tibial, collateral ligament attaches from the medial epicondyle of the femur to the medial aspect of the shaft of the tibia. This ligament becomes taut on extension of the knee, abduction of the tibia on the femur, and external rotation of the tibia on the femur. The medial collateral ligament provides some help in preventing anterior displacement of the tibia on the femur. The lateral, or fibular, collateral ligament attaches from the lateral epicondyle of the femur to the head of the fibula. This ligament becomes taut on extension, adduction, and external rotation of the tibia on the femur. The tendon of the biceps femoris almost completely covers the lateral collateral ligament, and the popliteus tendon runs beneath it and separates it from the meniscus.
Surrounding the external aspect of the joint is the fibrous joint capsule attaching at the margins of the articular cartilage. The inferior portion of the capsule has been referred to as the coronary ligament. A substantial thickening in the medial portion of the joint capsule has fibrous attachments to the periphery of the medial meniscus, thereby binding it firmly to the femur and loosely to the tibia. The lateral aspect of the joint capsule has a similar thickening and has fibrous attachments to the lateral meniscus.
The posterior fibers of the medial collateral ligament blend with the joint capsule and the attachments to the medial meniscus. The patellofemoral ligaments form as thickenings of the anterior joint capsule and extend from the middle of the patella to the medial and lateral femoral condyles. Their function is to stabilize the patella in the patellar groove. The posterior thickening of the joint capsule arches over the popliteus tendon, attaches to the base of the fibular head, and becomes taut in hyperextension. Also blending with the posterior joint capsule is the oblique popliteal ligament, which forms as an expansion of the semimembranosus tendon and runs obliquely in a superior and lateral direction to attach to the lateral femoral condyle. It also becomes tight in hyperextension.
Lying between the femur and the tibia are the two semilunar cartilages called menisci (Figure 6-168). The menisci are shaped so that the more peripheral portions are thicker than the central portion. This serves to deepen the articular surface on the tibial plateau and provide additional stability to the joint. Because they increase the surface area of the joint, the menisci help to share the load in weight-bearing across the joint by distributing weight over a broader area. They also aid in the lubrication and nutrition of the joint and, coupled with their shock-absorbing capabilities, help to decrease cartilage wear. The periphery of each meniscus attaches to the joint capsule, and the inner edge remains free. The medial meniscus is C-shaped, and the posterior portion is larger than the anterior. The anterior horn inserts on the intercondylar area of the tibia, and the posterior horn inserts just anterior to the attachment of the posterior cruciate ligament. The lateral meniscus is almost a complete circle, with the tips of each horn quite close to one another. The lateral meniscus is more mobile than the medial meniscus.
Stabilizing the knee are the many muscles that cross it (Table 6-16). Laterally, the iliotibial band attaches to the lateral condyle of the tibia, providing anterolateral reinforcement and stabilization against excessive internal rotation of the tibia on the femur. Crossing the anterior aspect of the knee is the quadriceps tendon. It is formed by the junction of the four heads of the quadriceps muscle, which consist of the vastus lateralis, the vastus medius, the vastus medialis, and the rectus femoris. The quadriceps musculature functions to extend the knee. Balanced activity between the vastus medialis and lateralis maintains optimal orientation of the patella within the patellofemoral groove. The sartorius and gracilis muscles provide medial stability to the joint. The sartorius also assists in knee and hip flexion, external rotation of the femur, and internal rotation of the tibia, depending on whether the extremity is weight-bearing. The gracilis acts to adduct the femur and assists in knee flexion and internal rotation of the tibia. Posteromedial reinforcement of the joint is supplied by the pes anserinus tendons (semitendinosus, gracilis, and sartorius), and the semimembranosus tendon. These help to prevent external rotation, abduction, and anterior displacement of the tibia. Posterolateral support from the biceps femoris tendon helps to check excessive internal rotation and anterior displacement of the tibia. The hamstring muscle is the primary knee flexor; the biceps femoris also provides some external rotation. Posterior reinforcement of the knee joint is provided by the gastrocnemius muscle and the popliteus muscle (Figure 6-169). The gastrocnemius is a primary ankle plantar flexor but also assists in knee flexion. The popliteus internally rotates and flexes the tibia when the limb is not bearing weight. The converse occurs when the limb is bearing weight.
TABLE 6-16 Actions of the Muscles of the Knee Joint
Action | Muscles |
---|---|
Extension | Quadriceps |
Flexion | Hamstrings, gracilis, sartorius, tensor fascia lata, and popliteus |
Internal rotation | Sartorius, gracilis, semitendinosus, semimembranosus, and popliteus |
External rotation | Biceps femoris and tensor fascia lata (iliotibial tract) |
Figure 6-169 Lateral (A) and medial (B) aspects of the right knee, demonstrating muscular attachments.
Because the knee is exposed to a variety of demands in human locomotion, numerous bursae are located in relationship to the knee joint and to the synovial cavity. The synovial membrane of the knee joint is the most extensive of any in the body. The suprapatellar pouch, or quadriceps bursa, is actually an extension of the synovial sac that runs from the superior aspect of the patella upward beneath the quadriceps tendon and then folds back on itself to form a pouch inserting on the distal femur above the condyles (Figure 6-170). The prepatellar bursa is relatively large but superficial and lies between the skin and the patella. It can become inflamed with prolonged kneeling activities. The deep and superficial infrapatellar bursae lie just under and over the patellar tendon, respectively. Bursal sacs also lie between the semimembranosus tendon and the medial head of the gastrocnemius muscle, and two bursae separate the mediolateral heads of the gastrocnemius muscle from the joint capsule. (A Baker cyst occurs with effusion of the medial gastrocnemius and the semimembranosus bursae.) A bursa also lies under the pes anserine tendon, separating it from the tibial collateral ligament.
The knee joint must provide a broad ROM while maintaining its stability. It must react to rotational forces, as well as absorb shock, and then immediately prepare for propulsion. The knee functions as a modified hinge joint, with flexion and extension being its primary motions. Limited rotation occurs, especially when the joint is not in the closed-packed position (extension) (Box 6-12). Flexion and extension of the knee are combinations of roll, slide, and spin movements, which effectively shift the axis of movement posteriorly as the knee moves from extension into flexion (Figure 6-171). Similar to elbow flexion, knee flexion is limited by soft tissue of the calf and posterior thigh, and extension is limited by the locking of the joint from bony and soft tissue elements in the joint’s close-packed position. Moreover, the so called screw-home mechanism, which is a combination of external rotation of the tibia occurring with knee extension, further approximates the osseous structures and tightens the ligamentous structures to stabilize the joint (Figure 6-172). The marked incongruent positions of the tibiofemoral joint are reduced by the fibrocartilaginous menisci. These also help to distribute the forces of compressive loading over a greater area and reduce compressive stresses to the joint surfaces of the knee. ROMs of the knee are listed in Table 6-17.
Figure 6-172 “Screw-home” mechanism of the knee, combining external rotation with extension, which maximally approximates the joint surfaces.
(Modified from Nordin M, Frankel VH: Basic biomechanics of the musculoskeletal system, ed 2, Philadelphia, 1989, Lea & Febiger.)
TABLE 6-17 Arthrokinematic and Osteokinematic Movements of the Knee Joint
Osteokinematic Movements | Degrees | Arthrokinematic Movements |
---|---|---|
Flexion | 130 | Roll and glide |
Extension | 10 | Roll and glide |
Internal rotation | 10 | Rotation |
External rotation | 10 | Rotation |
The patellofemoral joint plays an active role in flexion and extension of the knee joint. This joint has a gliding motion, moving caudal approximately 7 cm when going from full flexion to full extension. The articular surface of the patella never makes complete contact with the femoral condyles; the joint space decreases with flexion, and at full flexion, the patella sinks into the intracondylar groove (Figure 6-173). This characteristic is important in helping to increase weight-bearing capabilities in a flexed position (squatting). Therefore, the patella plays two important biomechanical roles for the knee. It primarily aids in knee extension by producing anterior displacement of the quadriceps tendon, thereby lengthening the lever arm of the quadriceps muscle force. In addition, it allows a wider distribution of compressive force on the femur, especially in a fully flexed position.
Figure 6-173 The relationship of the patella to the femur in extension (A) and flexion (B) of the knee.
The patella has an optimal position in relationship to the knee joint in both the vertical and sagittal planes. On a lateral view of the knee, the length of the patella is compared with the distance from the inferior pole of the patella to the tibial tubercle (Figure 6-174). If the difference of these two numbers is greater than 1 cm, the position of the patella is either high (alta) or low (baja), depending on which distance is greater. To determine if the patella is aligned properly in the femoral groove, the Q angle can be determined. After locating the center of the patella, extend a line up the center of the patellar tendon through the center point. Then draw a line from the center of the patella toward the ASIS (Figure 6-175). The resultant angle is the Q angle. The normal Q angle is 10 to 15 degrees, being slightly greater in females. Muscle imbalance and rotational disrelationships of the tibia and femur produce changes in the Q angle.
Figure 6-174 Identification of the position of the patella. If A–B is greater than 1 cm, a low (baja) patella exists; if B–A is greater than 1 cm, a high (alta) patella exists.
Figure 6-175 Q angle, the angle formed from the intersection of lines from the center of the patella to the anterior superior iliac spine and along the quadriceps tendon.
The superior tibiofibular joint is mechanically linked to the ankle, but a dysfunctional process in this joint presents clinically as pain in and about the knee. Therefore, inclusion of it in a discussion of the knee is clinically relevant. The superior tibiofibular joint allows superior and inferior movement, as well as internal and external rotation of the fibula. During ankle dorsiflexion the fibula internally rotates and rises superiorly. The addition of ankle eversion causes some posterior displacement of the fibular head. Ankle plantar flexion draws the fibula inferiorly and creates external rotation. The addition of ankle inversion draws the fibular head anteriorly.
The knee joint is affected by a wide variety of clinical syndromes. Trauma is by far the most common causative agent. Injuries to the collateral ligaments usually require some type of traumatic force. The mechanism of most medial collateral ligament injuries is a twisting external rotation strain while the knee is flexed or a valgus (abduction) blow to the knee. A lateral collateral ligament injury may occur with a varus (adduction) blow to the knee, and when internal rotation and hyperextension occur with this force, the fibular head may avulse.
The anterior cruciate ligament is commonly injured by forced internal rotation of the femur on a fixed tibia while the knee is abducted and flexed. The posterior cruciate ligament can be torn when a traumatic force is delivered to the front of the flexed tibia, driving it posteriorly under the femur. Forced external rotation of the femur on the tibia while the foot is fixed and the knee is abducted and flexed will also injure the posterior cruciate ligament.
After traumatic injuries to the knee causing ligamentous injury, joint instabilities are likely. The knee can undergo translational or rotational instabilities. Orthopedic stress tests are performed to identify the presence of joint instability.
The menisci are another site of possible injury. A trauma that couples rotation or violent extension of the knee may cause an isolated longitudinal or transverse tear in the meniscus.
Problems involving the patellofemoral joint complex are common and may be more frequent than ligamentous or meniscus disorders. A patient complaining of vague aching pain about the knee that is aggravated by going up or down stairs likely has patellofemoral joint dysfunction. Patellar tracking problems can occur primarily from injuries to the knee and quadriceps mechanism or secondarily in response to problems affecting the ankle or hip. Chondromalacia patella, an erosion and fragmentation of the subpatellar cartilage, may be secondary to trauma, recurrent subluxation, pronated feet, postural instability, short leg syndrome, or excessive femoral torsion with resultant irregular Q angle.
An avulsion fracture with resulting aseptic necrosis of the tibial tuberosity may occur from a sudden contraction of the quadriceps femoris. This condition is called Osgood-Schlatter’s disease and is more common in boys.
The knee joint is innervated by segments L3–S1, and therefore in cases of pain of nontraumatic onset, lesions situated elsewhere in segments L3–S2 must be ruled out. The lumbar spine, hip, and foot are sources of referred pain to the knee (Figure 6-176).
To begin the evaluation of the knee, observe the knee for evidence of swelling, asymmetry of contours, and postural changes (valgus, varus, or recurvatum). Movements of the knee during gait should be smooth and rhythmic, with the knee bent during the swing phase and fully extended at heel strike.
Identify osseous symmetry and pain production through static palpation of the tibial plateau, tibial tubercle, adductor tubercle, femoral condyles, femoral epicondyles, fibular head, patella, and trochlear groove (Figure 6-177). Determine the Q angle.
Figure 6-177 Patterns of referred pain to and from the knee.
(Modified from Magee DJ: Orthopedic physical assessment, ed 5, St Louis, 2008, Saunders.)
Identify tone, texture, and tenderness changes through soft tissue palpation of the quadriceps muscle, the infrapatellar tendon, the collateral ligaments, the pes anserine tendons, the peroneal nerve, the tibial nerve, the popliteal artery, the hamstring muscles, and the gastrocnemius and soleus muscles.
Evaluate accessory joint motions for the knee articulations to determine the presence of joint dysfunction (Table 6-18). Assess long-axis distraction with the patient supine and the affected leg slightly abducted. Stand and face the patient, straddling the affected leg so that your knees can grasp the patient’s distal leg just proximal to the malleoli. Use both hands to palpate the knee joint at its medial and lateral aspects and use your legs to create a long-axis distraction while palpating with your hands for a springy end feel (Figure 6-178). Alternatively, one hand may stabilize the patient’s femur on the table while the other hand palpates for end feel.
TABLE 6-18 Accessory Joint Movements of the Knee Joint
Joint | Movement |
---|---|
Tibiofemoral | |
Patellofemoral | |
Tibiofibular |
A-P, Anterior-to-posterior; I-S, inferior-to-superior; L-M, lateral-to-medial; M-L, medial-to-lateral; P-A, posterior-to-anterior; S-I, superior-to-inferior.
Evaluate A-P and P-A glide with the patient supine and the involved knee flexed to 90 degrees with the foot flat on the table. Either kneel or sit on the patient’s foot for stability while grasping the proximal tibia with both hands. Stress the proximal tibia in an A-P and P-A direction, looking for a springing end feel (Figure 6-179).
Figure 6-179 Assessment of anterior-to- posterior and posterior-to-anterior glide in flexion of the left tibiofemoral joint.
To evaluate internal and external rotation, use the same positions as for A-P and P-A glide. Stress the proximal tibia internally and externally to feel for a springing end feel (Figure 6-180).
Figure 6-180 Assessment of external and internal rotation in flexion of the left tibiofemoral joint.
Evaluate M-L and L-M glide with the patient supine and the involved leg abducted beyond the edge of the table. Then straddle the patient’s involved leg just proximal to the ankle while grasping the proximal tibia with both hands. Apply an M-L and L-M stress to the knee joint to identify a springing end feel. Alternatively, grasp the patient’s involved leg with the tibia held between your arm and body, with one hand on the tibia and one hand on the femur. The two hands can then create an M-L or L-M stress action (Figures 6-181 and 6-182).
Evaluate the patellofemoral articulation for M-L glide (Figure 6-183), L-M glide (Figure 6-184), S-I glide (Figure 6-185), and I-S glide (Figure 6-186), with the patient lying supine and the involved leg straight in passive knee extension. Contact the borders of the patella with both thumbs and apply a stress to the patella in M-L, L-M, S-I, and I-S directions, feeling for a comparative amount of movement from side to side, as well as a springing quality of movement.
Evaluate A-P and P-A glide of the tibiofibular articulation with the patient supine and the affected knee bent to 90 degrees and the foot flat on the table. Either kneel or sit on the patient’s foot to stabilize it and grasp the proximal fibula with the outside hand while stabilizing the proximal tibia with the inside hand. Then stress the fibula in P-A and A-P directions, looking for a springing end feel (Figures 6-187 and 6-188).
Perform I-S and S-I glide of the tibiofibular articulation with the patient supine, the affected leg straight, and the knee in the relaxed extension. Use a digital contact of the cephalic hand to palpate the proximal fibula while grasping the patient’s foot with your caudal hand. Then passively invert (with plantar flexion) and evert (with dorsiflexion) the patient’s ankle while palpating for the fibula to move it superiorly and inferiorly (Figures 6-189 and 6-190).
The manipulative techniques used to treat knee disorders aim to restore normal joint mechanics, which will then ideally allow full pain-free functioning of the knee joints. The three joints associated with the knee should be evaluated for characteristics of dysfunction when there are knee symptoms present. Box 6-13 identifies the adjustive procedures for the joints of the knee.
BOX 6-13 Knee Adjustive Techniques
Figure 6-193 Adjustment for external and internal rotation of the left tibiofemoral joint in the supine position.
The ankle and foot can be discussed together because they are intimate components of a very intricately functioning unit. Together they make up a significant component in a kinetic chain responsible for propulsion and balance. These joints may be viewed as initial supports for the musculoskeletal frame, because they form the base on which all other osseous and muscular mechanisms reside. To the contrary, these joints may also be viewed as a terminal segment, in that they must translate and carry out the messages from the central nervous system through the hip and knee. This joint complex must attenuate weight-bearing forces, support and propel the body, and maintain equilibrium.15 The feet and ankles must therefore provide the two paradoxical qualities of stability and pliability. They achieve these requirements through an interaction of interrelated joints, connective tissues, and muscles. Certainly, this part of the lower extremity is subject to a multiplicity of traumatic and postural disorders, leading to numerous joint dysfunction syndromes.
The distal tibia and fibula join with the talus to form a mortise-type hinge joint called the talocrural joint. The calcaneus, the largest tarsal bone, articulates with the talus, forming the subtalar joint. The navicular articulates with the talus proximally and cuneiforms distally. The cuboid articulates with the calcaneus proximally and with the fourth and fifth metatarsals distally (Figure 6-204). It also articulates with the navicular and third cuneiform medially. The first, or medial, cuneiform articulates with the first metatarsal; the second, or intermediate, cuneiform articulates with the second metatarsal; and the third, or lateral, cuneiform articulates with the third metatarsal. Two phalanges complete the structure of the great toe, and three phalanges complete the bony structures of each of the other four toes. The function of the tibia is to transmit most of the body weight to and from the foot, and although the fibula plays a very important role in ankle stability, it is not directly involved in the transmission of weight-bearing forces. Functionally, the talus serves as a link between the leg and the foot.
Although many ligaments and joint capsules are associated with the foot and the ankle, some are more important to localize, palpate, and functionally understand (Figures 6-205, 6-206 to 6-207). The deltoid ligament, composed of four parts, provides medial stability to the ankle by attaching from the medial malleolus to the talus anteriorly and posteriorly, as well as to the navicular and calcaneus. Laterally, the ankle is secondarily stabilized by five fibular ligaments: the anterior and posterior tibiofibular ligaments, the anterior and posterior talofibular ligaments, and the calcaneofibular ligament. The plantar calcaneonavicular (spring) ligament attaches from the sustentaculum tali to the navicular (Figure 6-208). The function of this ligament is to keep the medial aspect of the forefoot and hindfoot in apposition and, in so doing, help to maintain the arched configuration of the foot.
Similar to the way the muscles of the wrist are located in the arm, the muscles of the ankle are located in the calf. Posteriorly, the large calf muscle group (gastrocnemius and soleus) attaches from the femoral condyles, proximal fibula, and tibia to the calcaneus, providing plantar flexion of the ankle. The tendon of the tibialis posterior passes under the medial malleolus to attach to the plantar surfaces of the navicular; cuneiforms; the cuboid; and the second, third, and fourth metatarsals. As such, it serves primarily as an inverter and adductor, or supinator, of the foot. The flexor hallucis longus and flexor digitorum longus muscles also have tendons that pass under the medial malleolus, with each inserting on the distal phalanx of each toe, thereby creating flexion of the toes. Anteriorly are the extensor digitorum longus, tibialis anterior, and extensor hallucis longus muscles. The extensor digitorum longus attaches to the dorsal surfaces of each of the distal phalanges, primarily extending the four toes, but also serving to dorsiflex, evert, and abduct the foot. The extensor hallucis longus is the primary extensor of the big toe. The tendon of the tibialis anterior passes over the ankle joint and across the medial side of the dorsum of the foot, and inserts into the medial and plantar surface of the medial cuneiform bone and the base of the first metatarsal. It functions as the primary dorsiflexor of the ankle, but because of its insertion, it also inverts and adducts the foot. On the lateral aspect is the peroneus muscle group. The tendons of the peroneus longus and brevis pass under the lateral malleolus and cross the cuboid to insert into the medial cuneiform and base of the first metatarsal. The chief action, then, of both peronei muscles is to evert the foot. The peroneus tertius is continuous, with the origin of the extensor digitorum longus muscle; its tendon diverges laterally to insert into the dorsal surface of the base of the fifth metatarsal bone. It works with the extensor digitorum longus to dorsiflex, evert, and abduct the foot.
The ankle musculature can be divided into positional groups and divided according to the actions they perform (Table 6-19). The gastrocnemius, soleus, and plantaris muscles lie posteriorly and are responsible for plantar flexion of the foot and ankle. The extensor hallucis longus, extensor digitorum longus, peroneus tertius, and tibialis anterior muscles lie anteriorly and serve primarily to dorsiflex the foot and extend the toes. The peroneus longus and brevis muscles are situated laterally and pronate and evert the foot. The tibialis posterior, flexor digitorum longus, and flexor hallucis longus muscles are medial and function to invert the foot and flex the toes. The intrinsic muscles of the foot lie in layers and generally perform the actions indicated by the muscle names.
TABLE 6-19 Actions of the Muscles of the Foot and Ankle
Action | Muscles |
---|---|
Ankle plantar flexion | Gastrocnemius, soleus, and plantaris |
Ankle dorsiflexion | Extensor digitorum longus, tibialis anterior, peroneus tertius, and extensor hallucis longus |
Ankle inversion (adduction) | Tibialis posterior and tibialis anterior |
Ankle eversion (abduction) | Extensor digitorum longus, peroneus longus and brevis, and peroneus tertius |
Toe flexion | Flexor digitorum longus and flexor hallucis longus |
Toe extension | Extensor digitorum longus and extensor hallucis longus |
The functional biomechanics of the foot and ankle must include the ability to bear weight and allow flexible locomotion. The ankle joint is a uniplanar hinge joint, with talus motion occurring primarily in the sagittal plane about a transverse axis. The lateral side of the transverse axis is skewed posteriorly from the frontal plane (Figure 6-209). The primary movement at the ankle mortise is dorsiflexion (20 to 30 degrees) and plantar flexion (30 to 50 degrees) (Table 6-20). Through the normal gait pattern, however, only 10 degrees of dorsiflexion and 20 degrees of plantar flexion are required.24
Figure 6-209 Dorsiflexion and plantar flexion in the foot and ankle occur around an oblique axis, and eversion and inversion occur around the longitudinal axis.
(Modified from Wadsworth CT: Manual examination and treatment of the spine and extremities, Baltimore, 1988, Williams & Wilkins.)
TABLE 6-20 Arthrokinematic and Osteokinematic Movements of the Ankle and Foot Joints
Osteokinematic Movements | Degrees | Arthrokinematic Movements |
---|---|---|
Ankle dorsiflexion | 20 | Roll and glide |
Ankle plantar flexion | 50 | Roll and glide |
Subtalar inversion | 5 | Roll and glide |
Subtalar eversion | 5 | Roll and glide |
Forefoot abduction | 10 | Roll and glide |
Forefoot abduction | 10 | Roll and glide |
The subtalar joint formed between the talus and calcaneus is also a hingelike joint. The axis of movement passes through all three cardinal planes of movement, thereby allowing movement to some extent in all three planes. Movement of this joint, then, includes the complex movement of supination and pronation of the calcaneus under the talus. Supination is a combination of inversion, adduction, and plantar flexion, whereas pronation is a combination of eversion, abduction, and dorsiflexion (Figure 6-210). The subtalar joint has the important function of absorbing shock at heel strike and rotating the lower extremity in the transverse plane during the stance phase of gait.
Figure 6-210 Pronation and supination movements in free swing action (open kinetic chain) (A) and with weight-bearing (closed kinetic chain) (B).
The transverse or midtarsal joint is composed of the talonavicular and calcaneal cuboid articulations. The amount of movement occurring at these joints, which function in unison, depends on whether the foot is in pronation or supination. The supinated position creates a divergence of the axes of movement, which decreases the amount of movement, setting up a rigid bony structure necessary for stability at the heel-strike stage of gait. Pronation, on the other hand, allows for the axes of motion to become aligned, which allows for increased mobility and decreased stability. The ligaments that run on the plantar surface between these tarsal bones are an essential component for absorbing stress and maintaining the longitudinal arch. Table 6-21 identifies the close-packed and loose-packed positions for the ankle and toe joints.
TABLE 6-21 Close-Packed and Loose-Packed (Rest) Positions for the Ankle and Foot Joints
Joint | Close-Packed Position | Loose-Packed Position |
---|---|---|
Tibiotalar articulation | Full dorsiflexion | 10 degrees of plantar flexion midway between full inversion and eversion |
Toes | Full extension | Flexion |
The individual tarsal joints, metatarsal-tarsal joints, metatarsophalangeal joints, and interphalangeal joints enhance the foot’s stability or flexibility. They must provide a base for the stance phase, as well as the necessary hinges for flexion and extension during toe-off. Ligaments and tendons further enhance stability and flexibility by maintaining arches in the foot (Figure 6-211).
During the gait cycle, two main phases are described. The first occurs when the foot is on the ground and is called the stance phase, and the second occurs when the foot is not contacting the ground and is called the swing phase (Figure 6-212). The stance phase is further divided into a contact component, midstance component, and a propulsive component. Movements of the leg and foot change through the different components. Pronation of the subtalar joint occurs initially at heel strike while the tibia internally rotates. This is followed by supination of the subtalar joint and external rotation of the tibia through midstance and propulsive stages, as well as through the swing phase of gait. This creates a shifting area of weight-bearing across the foot, starting from the posterolateral aspect of the calcaneus and curving over to the first metatarsophalangeal joint. Abnormal supination or pronation of the subtalar joint will result in altered gait patterns, as well as weight-bearing stresses on the plantar surface of the foot.
With the concentrated stress that occurs to the foot and ankle during bipedal static and dynamic postures, these areas are susceptible to many injuries. Commonly, ankle injuries have an acute traumatic onset, whereas the foot is more likely to develop chronic and insidious onset disorders from stress overload. Pain and paresthesias arising from the lower lumbar or first sacral NRs should not be overlooked (Figure 6-213). Most foot and ankle pain, however, arises from local disease or pathomechanic processes.
The most common traumatic injury to this area is the inversion sprain of the ankle, causing a separation to the lateral compartment with damage to the anterior talofibular ligament and possibly to the calcaneofibular ligament. Rarely does inversion occur alone, because usually plantar flexion of the ankle, as well as external rotation of the leg, also occur.
An eversion sprain involving trauma to the medial aspect of the ankle and affecting the deltoid ligament usually occurs when the foot is fixed in an excessive amount of pronation and the individual turns forcefully toward the opposite foot. The stress is applied first to the anterior tibiofibular ligament.
Shin splints refer to a generalized, deep aching or, sometimes, sharp pain along the tibia. It is considered an overuse or abuse syndrome occurring commonly because of running or jumping on a hard surface. This activity causes the talus to be driven upward into the mortise, forcing the tibia and fibula to separate. Stress to the interosseous membrane results and may cause a periostitis. Furthermore, activity of the anterior tibialis may result in fluid becoming entrapped within the fascial covering, creating a compartment syndrome.
Plantar fasciitis results as a strain to the plantar fascia on the sole of the foot. This may be a result of standing on hard surfaces, quick acceleration or deceleration, repeated shocks, standing on ladders, or long periods of pronation. A calcaneal heel spur may eventually occur as the fasciitis continues or worsens. The fascia will pull the periosteum off the calcaneus, creating a painful periostitis, and bone will be laid down at the site of stress.
Hallux valgus is a lateral deviation of the big toe, usually with a concomitant metatarsal varum. Improperly fitting footwear, as well as an unstable and pronated foot, has been blamed for this condition.
The evaluation of the foot and ankle begins with observation during static posture, as well as gait for symmetry, arches, toe deformities, and soft tissue swelling. Inspect the plantar surface of the foot for signs of weight-bearing asymmetry in the form of callous formation. Identify osseous symmetry and pain production through static palpation of the distal tibia and fibula (malleoli), dome of the talus, navicular, cuboid, calcaneus, cuneiforms, metatarsals, and phalanges.
Identify tone, texture, and tenderness changes through soft tissue palpation of the medial and lateral ligaments, Achilles tendon, and plantar fascia, as well as the musculature that controls movement of the foot and ankle. Additionally, palpate the posterior tibial artery and dorsalis pedis artery.
Evaluate accessory joint movements for the foot and ankle articulations to determine the presence of joint dysfunction (Table 6-22). Assess long-axis distraction of the tibiotalar articulation or ankle mortise joint with the patient supine, the knee flexed to approximately 90 degrees, and the hip flexed and abducted. Sit on the table between the patient’s legs and face caudal. Place web contacts over the dome of the talus and superior aspect of the calcaneus, applying a distraction force through both hands (Figure 6-214).
TABLE 6-22 Accessory Joint Movements of the Foot and Ankle Joints
Joint | Movement |
---|---|
Tibiotalar joint | |
Subtalar joint | |
Tarsals (cuboid, navicular, and cuneiforms) | |
Intermetatarsal joints | |
Metatarsophalangeal and interphalangeal joints |
A-P, Anterior-to-posterior; L-M, lateral-to-medial; M-L, medial-to-lateral; P-A, posterior-to-anterior.
Figure 6-214 Patterns of referred pain to and from the ankle.
(Modified from Magee DJ: Orthopedic physical assessment, ed 5, St Louis, 2008, Saunders.)
Evaluate A-P and P-A glide of the ankle mortise joint with the patient supine and the hip and knee both slightly flexed so that the calcaneus rests on the table. Stand at the side of the table and place a web contact of your cephalic hand over the anterior aspect of the distal tibia while placing a web contact with your caudal hand over the anterior aspect of the dome of the talus. With both hands, grasp the respective structures and maintain the joint in its neutral position. Apply an A-P and P-A translational force through both hands, working in opposite directions, looking for a springing joint play movement (Figure 6-215).
Figure 6-215 Assessment of anterior-to- posterior and posterior-to-anterior glide of the right tibiotalar joint.
Assess M-L and L-M glide of the tibiotalar articulation with the patient supine. Stand at the foot of the table, facing cephalad. Grasp the dome of the talus with the fingers of both hands, using the thumbs to grasp under the plantar surface of the foot. Then stress the talus in an M-L and L-M direction, feeling for a springing joint play movement (Figure 6-216).
Figure 6-216 Assessment of medial-to-lateral (inversion) (A) and lateral-to-medial (eversion) (B) glide of the left tibiotalar joint.
Evaluate subtalar joint glide with the patient lying in the prone position and the knee flexed to approximately 60 degrees. Stand at the foot of the table, facing cephalad, with the plantar surface of the patient’s toes resting against your abdomen. Then grasp the calcaneus with palmar contacts while interlacing your fingers in a “praying-hands” position. Use both hands to create A-P and P-A glide, as well as M-L and L-M glide movements (Figure 6-217).
Figure 6-217 Assessment of anterior-to-posterior, posterior-to-anterior, medial-to-lateral, and lateral-to-medial glide of the right subtalar joint.
Perform A-P and P-A glide of the navicular, cuboid, and cuneiforms by grasping the specific tarsal bone while stabilizing the proximal tarsal and creating an A-P and P-A glide movement (Figure 6-218).
Figure 6-218 Assessment of anterior-to-posterior and posterior-to-anterior glide of the left cuboid (same procedure used for the navicular and cuneiforms).
Perform A-P and P-A shear of the intermetatarsals by grasping adjacent metatarsals with each hand and creating an A-P and P-A shear (Figure 6-219).
Figure 6-219 Assessment of anterior-to-posterior and posterior-to-anterior shear between the left metatarsals.
Evaluate the metatarsophalangeal and interphalangeal joints for A-P and P-A glide, M-L and L-M glide, axial rotation, and long-axis distraction by grasping the metatarsals with one hand for stabilization and placing the specific phalanx between the index and middle fingers of the other hand (Figure 6-220).
The manipulative techniques used to treat ankle and foot disorders aim to restore normal joint mechanics, which will then ideally allow full pain-free functioning of ankle and foot hip joints. Box 6-14 identifies the adjustive procedures for the ankle and foot.
BOX 6-14 Ankle and Foot Adjustive Techniques
Figure 6-238 Adjustment for internal and external rotation and anterior-to-posterior, posterior-to-anterior, lateral-to-medial, and medial-to-lateral glide of the right metatarsophalangeal joints (same procedure for the interphalangeal joints).
The use of manipulative or adjustive techniques with peripheral joint problems is a valuable aspect in chiropractic practice, requiring no more or no less skill than techniques for the spine.